Wednesday, February 19, 2020

Three stages in the international product life cycle theory Essay

Three stages in the international product life cycle theory - Essay Example Production of a particular product in a certain region is based on market analysis regarding resource availability, needs, and purchasing power of the target market. The characteristics of this stage are that the sales volumes and prices are high. At this level, the demand is greater than the supply with little competitors in the niche thus allowing for expansion of the business. Furthermore, at this stage the advertisement is at its peak with the technique having tremendous effect on the target market. During the growth stage, international imitators have had no chance to create imitations at a cheaper price that eventually destabilizes the market. A good example of a company that vividly elaborates this stage is Chinese Tecno Mobile Phone Company. The company based in the most populated country came up with a product to meet the demand for cheap Smartphone. The company realized massive profits in 2004 when it started. The next stage of the cycle is the maturity stage. In this stage, the demand becomes level, and the rate of increase of sales is reduced. At this level, the imitators have had enough time to create a product that creates competition in the available market at a cheaper price. However, esteemed customers who enjoy services of the original product which explained a level demand of the product as no new customers are using the product. Producer of the original product at this stage might opt to reduce prices if competition on the available market becomes stiff. The decreased sales volume and prices lead to a reduction in the amount of profits realized at a specific financial period (Funk,  2004). A good example of this is the Antex Knitting Meals located in Los Angeles. The profits realized by the company after establishment in 1979 were high compared to the current profits selling 2.7 million yards of fabric per week. Currently, the company sales add up to 1.5million yards of fabric per week. Decline is the

Tuesday, February 4, 2020

High Fiber diet and regulation of oncogenes Research Paper

High Fiber diet and regulation of oncogenes - Research Paper Example Statistics reflecting the estimated cases of cancer and deaths caused by cancer in the United States have also been taken into account, so as to further strengthen the relevance of the topic. Oncogenes are proto-oncogenes turned bad -- genes that have undergone mutations (American Cancer Society, 2011); mutations are changes in the makeup of a gene’s building blocks, DNA (American Cancer Society, 2011). According to the theory of gene mutation, carcinogen interaction with deoxyribonucleic acid leads to irrevocable genetic changes (Devi, 2005, p. 18). Even a minute alteration in the arrangement of these building blocks can create a strong negative impact (American Cancer Society, 2011). Normally, proto-oncogenes are responsible for the encoding of proteins, which promote cell division and impede cell differentiation (Chial, 2008). On the other hand, these mutated proto-oncogenes â€Å"typically exhibit increased production of these proteins, thus leading to increased cell division, decreased cell differentiation, and inhibition of cell death† (Chial, 2008). The aforementioned process leads to cancer (Chial, 2008); hence, there is a need to regulate these onc ogenes. A â€Å"diet high in fiber† is made known to have a considerable impact in the shrinking of predisposition to â€Å"colorectal cancer† (Doheny, 2011). Doll and Peto were the first to highlight the existence of a link between diet composition and cancer -- an example of which is high-fiber diet and its effectivity in chemoprevention (as cited in Devi, 2005, p. 21). Carcinogenesis begins with initiation as normal genes are exposed to carcinogens (Devi, 2005, p. 16). After which, promotion follows -- a stage wherein the gene is repeatedly exposed to stimuli (Devi, 2005, p. 17). Thereafter, progression occurs; this is the proliferation of the mutated gene (Devi, 2005, p. 17). Devi